lipid almashinuvi buzilishi

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patofiziologiya ateroskleroza lipid almashinuvi buzilishi. t.f.n. raxmatova sanobar nizamovna * labda distrofiya charchoq semirish ateroskleroz arterioskleroz dislipo- proteinemiya darajasiga qarab lipid metabolizmining buzilishi klinik ko'rinishga qarab buzilishlar ovqat hazm qilish va assimilyatsiya buzish transmembrana olib yurmoq buzilishlar metabolizm hujayralardagi lipidlar (parenximasi) lipidozlar) 4 lipid almashinuvi buzilishi lipidozlar lipidozlar (grech. lipos –yog ', os – patologik jarayon) lipid metabolizmi patologiyasining tipik shakli: lipid metabolizmining buzilishi bilan tavsiflangan hujayralardagi (parenximali lipidozlar) yog 'to'qimalarida (semirish, charchoq, lipodistrofiya) qonda (dislipidemiya) arteriyalar devorlarida (ateroskleroz, arterioskleroz) * haddan tashqari (patologik) tanadagi yog 'to'planishi triglitseridlar shaklida * tana vaznining ortishi bilan odatdagidan 20-30% yuqori. * * * * tarqalishi: * xavf omillari: - ibs ( > 1,5 marta) - gipertenziya (> 2 marta)bolezni (> 2 raza) - neoplazmalar (?) © p.f.litvitskiy, 2004 © geotar-med, 2004 semirish evropa - aholining 20-60%.d.y rossiya - 60% dan yuqori. - ateroskleroz (> 2 marta)(> 2 raza) - qandli diabet (> 4 …

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adipotsitlar (neyropeptid y - leptin) ikkinchi - kaloriyali ovqatlar - katabolizmning intensivligini pasaytirish - tananing kam energiya iste'moli * leptin ta'sirining etishmasligi: · o'sayotgan ochlik l e p t i n p e p t i d e p t i d ( oq yog 'to'qimalarining adipotsitlari podavlyaet sintez i videlenie v krov neyropeptida y neyronami gipotalamusa massasi » 16 k da · semirish © p.f.litvitskiy, 2004 © geotar-med, 2004 · o'sayotgan ochlik · semirib ketish n e y r o p e p t y d y neuromediator ( orqa lateral ventral yadro – gipotalamus © p.f.litvitskiy, 2004 © geotar-med, 2004 * y neyropeptidi ta'sirining ortishi: ochlikni rag'batlantiradi, ishtahani oshiradi semirib ketish tana vazninig indeksi oddiy tvi = 20.0 – 24.9 ortiqcha vazn= 25.0 – 26.9 i semizlik darajasii = 27.0 - 30.0 ii semizlik darajasi = 30.0 - 40.0 iii semizlik darajasi- bolee 40.0 tvi = tana vazni(kg) …

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nizmi neyrotransmitter - peptid y sintezi va sekretsiyasining oshishi ochlikni rag'batlantirish, ishtahani oshirish ortiqcha ovlash semirish gipotalamik retseptorlarning y peptid sintez inhibitörlerine (leptin va boshqalar) hiposensitizatsiyasi + gipotalamik neyronlarning shikastlanishi semirib ketishning asosiy endokrin mexanizmlari mutlaq yoki nisbiyleptin etishmovchiligi qalqonsimon gormonlar ta'sirining yetishmasligi glyukokortikoidlarning ortiqcha ta'siri adipotsit insulin retseptorlari sonining va / yoki yuqori sezuvchanligining oshishi semirish glyukoza adipotsitlarga kiradi ortiqcha ovqatlanish lipoliz intensivligining pasayishi tananing energiya sarfini kamaytirish hujayralardagi glikogenolizning faollashishi dipotsit lipogenezining faollashishi leptin hipotiroid buyrak usti bezi insulin lipolizni inhibe qilish hissiyotning kuchayishi ochlik semirib ketishning metabolik mexanizmi ochlikni rag'batlantirish, ishtahani oshirish ortiqcha ovlash semirish mexanizmni buzish: "glikogenolizni giperlipidemiyada inhibe qilish" semizlik asorati e’tiboringiz uchun raxmat unknown-0.psd unknown-1.psd unknown-2.psd

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patofiziologiya ateroskleroza lipid almashinuvi buzilishi. t.f.n. raxmatova sanobar nizamovna * labda distrofiya charchoq semirish ateroskleroz arterioskleroz dislipo- proteinemiya darajasiga qarab lipid metabolizmining buzilishi klinik ko'rinishga qarab buzilishlar ovqat hazm qilish va assimilyatsiya buzish transmembrana olib yurmoq buzilishlar metabolizm hujayralardagi lipidlar (parenximasi) lipidozlar) 4 lipid almashinuvi buzilishi lipidozlar lipidozlar (grech. lipos –yog ', os – patologik jarayon) lipid metabolizmi patologiyasining tipik shakli: lipid metabolizmining buzilishi bilan tavsiflangan hujayralardagi (parenximali lipidozlar) yog 'to'qimalarida (semirish, charchoq, lipodistrofiya) qonda (dislipidemiya) arteriyalar devorlarida (ateroskleroz, arterioskleroz) * haddan tashqari (p...

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