revmatoid artritpatogenezi

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zagolovok slayda otsutstvuet revmatoid artrit biriktiruvchi to'qimaning surunkali tizimli kasaligi bo'lib, aksariyat periferik bo'g'imlarning eroziv-destruktiv poliartrit tipida avj oluvchi jaroxatlanishi bilan kechadi revmatoid artrit patogenezi etiologik omillar genetik moyillik t i v - limfotsitlar faoliyati disbalansi immun javob boshqarilishining buzilishi t- limfotsitlar defitsiti va antitanalar sintezi nazoratining etishmasligi igg bilan agregatsiyalangan β - limfotsitlar sintezi ig ro ga agregatsiyalangan antitanalar hosil bo'lishi ig va ro o'zaro ta'siri immun komplekslar hosil bo'lishi qon ivishi faol tizimi komppement komponentlari faollashuvi limfotsitlardan limfokinlar hosil bo'lishi xemotaksis (bo'g'im bo'shlig'iga neytrofillar oqib kelishi hujayralar zararlanishi yallig'lanish, sinovit, bo'g'imdan tashqari simptomlar rivojlanishi yallig'lanish mediatorlari ajralishi revmatoid artiritning tashxis mezonlari (a.r.a) 1. oxirgi 6 xafta davomida 1 soat davom etuvchi ertalabki karaxtlik 2. oxirgi 6 xafta davomida 3 yoki undan ortiq bo'g'imlarda shish 3. oxirgi 6 xafta va undan ko'p vaqt davomida kaft-barmoq va proksimal falangalararo bo'g'imlarda shish 4. bo'g'imlar jaroxatlanishining simmetrikligi 5. bo'g'imlarda tipik rentgenologik o'zgarishlar …

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'zgarishlar 2-globulinlar miqdori oshishi echt mm/soat s-reaktiv oqsil yo'q yo'q yo'q 15 >40 + + + va yuqori revmatoid artrit bosqichlari (rentgen ko'rinishi bo'yicha) i bosqich- osteoporoz, destruktiv rentgenologik o'zgarishlarsiz ii bosqich- tog'ay va suyakning bir oz jaroxatlanishi, bo'g'im oralig'ining bir oz torayishi, suyakda yakka eroziyalar iii bosqich - tog'ay va suyakning jaroxatlanishi, bo'g'im oralig'ining ifodalangan torayishi, ko'p sonli eroziyalar, bo'g'imning chala chiqishi iv bosqich - iii bosqich simptomlarining ankiloz bilan qo'shilib kelishi 0-yo'q i-professional mehnatga layoqati cheklangan ii-professional mehnatga layoqati yo'qolgan. iii-o'ziga xizmat qilish layoqati yo'qolgan revmatoid artrit – o'tkir boshlanishi, nisbatan tez avj olishi, ertalabki karaxtlik, bo'g'imlarning simmetrik jarohatlanishi, maxalliy yallig'lanish faolligi yuqoriligi, yallig'lanish xarakteridagi og'riqlar, revmatoid tugunchalar, ro aniqlanishi, o'ziga xos rentgenologik manzara o'tkir revmatik isitma – bo'g'im sindromi uchuvchan xarakterga ega, yirik bo'g'imlar jarohatlanishi, revmokardit belgilari ankilozlovchi spondiloartrit – bo'g'imlarning asimmetrik jarohatlanishi, umurtqa jarohatlanishi, hla b27 antigeni topilishi reyter sindromi – oyoqlar bo'g'imlarining surunkali artriti, tovon …

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ari - auronofin, ridaura, tauredon 3) d- penitsilamin - kuprenil 5) immunodepressantlar metotreksat arava - tsiklofosfamid 6) sulfanilamidlar - sulfosalazin - salazopiridin 7) remikeyd 8) leflyunomid ii - gormonoterapiya: 1) tizimli prednizolon, metipred 2) lokal kenalog, diprospan bo'g'im ichiga iii - n. p. v. p. tsog-2 ingibitorlari - ketonal - nimesil - brufen - ortofen iv – fizioterapiya muolajalari v – sanator-kurort davo pebmatou aptput wsiwwanionwa, nt as pebmmatoug aptput pa tachudy uammyhonofuk tabcugu kacaninuk keyualluiu taah4-xxxapakatt annapatahuh® cbyhkumohan c@talumobuunutu ahtu tachu®d (poccua pesmatonornap accouvaynacu nnehnym maxknucuga 30 cehtaop 2007 uv kabyn kunuhrah.) 1. acocun tawxuc: pesmatonug aptput ceponosuttus (m05.8) pesmatonug aptput cepoueratus (m06.0) pesmatonug aptput anoxvuga knvhwk waknnnapu: - mentu cungpomn (m05.0) - katranapga puboxknahagurah ctunn kacannurn (m06.1) pesmatong aptput axtumonnurn (m05.9, m06.4, m06.9) 2. knunnk gocknyunapn: - kyga apta socknun: kacannuk qabomunnur 1 vnn - key 6ockuun: kacannuk gabomuunurn 2 uwnn ba yhgah optnk + manga ba “npnk …

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“nknwnap iv - lll gockny + cyxak ahkuno3n 6. kywimmya ummyhonormk xapaktepunctuka - ahtulivtpyynnuh ahtutennanap: alilin - nosuttub auln - heratub 7. dyyhklnyohaan cuhd: |-— tynuk cakknahrah: y3-y3ura xu3mat kvm, npocbeccnohan oynmarah u npocdeccuonan mexxhatra naekatnuk ll - caknahrah: y3-y3ura xu3mat kynimlw, npocbeccnohan mexhattra naekatnuk; 4eknahrah: npoceccnonan oynmarah mexhatra naekatnuk lll - cakknahrah: y3-y3ura xu3mat kvm; yerapanahrah: npocdbeccnuonan oynmarah ba npocbeccuvohan mexhatra naekatnuk iv - yerapananhrah: y3-y3ura xu“3mat kyunulu, npocdbeccuohan oynmarah u npoceccuonan mexhattra naekatnuk . acopatnapn: . wkkunamun cuctem ammunongo3 . wkkunaamyn aptpo3 . octeeonopo3s (cuctem) . octeohekpo3 . tyhhen cuhapomnnap (kapnan kahan cuhgzpomy, tupccak hepby ba n. tibialis hu oocunuw cuhgapomnapn) 6. atnahto-akcuvan oyfum apmm 4nknwh, muenonatua ounah oupra, ymmyptka norohacuhn oymnh kncmmhn hoctaounnurn 7. atepocknepo3 ahwdn = 0 tupcak 6yfummu coxacuga tepu octm pebmatonug tyryhyanapu “oxkyww oy“hn” kypvuhuwugarn aecopmayna pahuhr oxupru gockuunapnura xoc 6ynrah ppehtrehoonnormk y3rapuwinap: kyn cohnn 3po3suanap, anhukca o6k nahkanapu guctan oyfumnapuga ucbogananhran, typfyh decbopmayna …

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About "revmatoid artritpatogenezi"

zagolovok slayda otsutstvuet revmatoid artrit biriktiruvchi to'qimaning surunkali tizimli kasaligi bo'lib, aksariyat periferik bo'g'imlarning eroziv-destruktiv poliartrit tipida avj oluvchi jaroxatlanishi bilan kechadi revmatoid artrit patogenezi etiologik omillar genetik moyillik t i v - limfotsitlar faoliyati disbalansi immun javob boshqarilishining buzilishi t- limfotsitlar defitsiti va antitanalar sintezi nazoratining etishmasligi igg bilan agregatsiyalangan β - limfotsitlar sintezi ig ro ga agregatsiyalangan antitanalar hosil bo'lishi ig va ro o'zaro ta'siri immun komplekslar hosil bo'lishi qon ivishi faol tizimi komppement komponentlari faollashuvi limfotsitlardan limfokinlar hosil bo'lishi xemotaksis (bo'g'im bo'shlig'iga neytrofillar oqib kelishi hujayralar zararlanishi yallig'lani...

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